LGI1 Dysfunction in Inherited and Acquired Epileptic Disorders
Contemporary Clinical Neuroscience, ISSN: 2627-5341, Page: 35-45
2015
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Book Chapter Description
LGI1 is a multifunctional brain protein whose dysfunction is related to several neurologic disorders as diverse as autosomal dominant lateral temporal epilepsy (ADLTE), autoimmune limbic encephalitis (LE), and glioma tumor progression. ADLTE is a genetic focal epilepsy characterized by auditory or aphasic aura and onset in infancy/adolescence, whereas autoimmune LE occurs in adult life and is characterized by amnesia, confusion, and seizures. The complex molecular mechanisms underlying these epileptic conditions are largely unknown. In this chapter, I outline the clinical features, the genetic or autoimmune causes, and a molecular mechanism possibly underlying both ADLTE and autoimmune LE.
Bibliographic Details
http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=85145894757&origin=inward; http://dx.doi.org/10.1007/978-3-319-12283-0_3; http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=84943384737&origin=inward; https://link.springer.com/10.1007/978-3-319-12283-0_3; https://dx.doi.org/10.1007/978-3-319-12283-0_3; https://link.springer.com/chapter/10.1007/978-3-319-12283-0_3
Springer Science and Business Media LLC
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