Less than 50% of variation in HDL cholesterol between and within individuals, is explained by established predictors
Atherosclerosis, ISSN: 0021-9150, Vol: 184, Issue: 1, Page: 178-187
2006
- 9Citations
- 22Captures
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Metrics Details
- Citations9
- Citation Indexes9
- CrossRef7
- Captures22
- Readers22
- 22
Article Description
Variation in serum high density lipoprotein cholesterol (HDL-C) concentrations is believed to be largely explained by triglycerides, but this has been mainly explored in cross-sectional analyses. Eight hundred and eighty one white male participants in a health screening program attended on a total of 2158 occasions for measurements that included fasting HDL-C, low density lipoprotein cholesterol (LDL-C) and homeostasis model assessment insulin resistance (HOMA-R). Baseline, change between-visit and repeated-measures regression models were used to analyse predictors of between- and within-individual variation in HDL-C. Independent predictors of between-individual variation included serum triglycerides (20.3 or 19.6% of the variance explained, depending on the model used), body mass index (BMI: 4.7 and 4.3%), cigarette smoking (3.3 and 1.5%) and alcohol consumption (0.4 and 1.1%). Within-individual variation in HDL-C was explained by changes in serum triglycerides (4.7 and 7.5%) and BMI (5.3 and 2.9%). In multivariate models, 24.3 and 24.9% of between-individual variation in HDL-C, and 7.9 and 8.8% of within-individual variation could be explained, depending on the model used. Sixty percent of the variation in HDL-C was due to unobserved factors. The majority of variation in HDL-C remains to be explained by influences other than the conventional variables: triglyceride and LDL cholesterol concentrations, insulin resistance, smoking and alcohol.
Bibliographic Details
http://www.sciencedirect.com/science/article/pii/S0021915005002583; http://dx.doi.org/10.1016/j.atherosclerosis.2005.03.032; http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=28344450867&origin=inward; http://www.ncbi.nlm.nih.gov/pubmed/15907857; https://linkinghub.elsevier.com/retrieve/pii/S0021915005002583; https://dx.doi.org/10.1016/j.atherosclerosis.2005.03.032
Elsevier BV
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