Establishment of a Duchenne muscular dystrophy patient-derived induced pluripotent stem cell line carrying a deletion of exons 51–53 of the dystrophin gene (CCMi003-A)
Stem Cell Research, ISSN: 1873-5061, Vol: 40, Page: 101544
2019
- 3Citations
- 17Captures
Metric Options: CountsSelecting the 1-year or 3-year option will change the metrics count to percentiles, illustrating how an article or review compares to other articles or reviews within the selected time period in the same journal. Selecting the 1-year option compares the metrics against other articles/reviews that were also published in the same calendar year. Selecting the 3-year option compares the metrics against other articles/reviews that were also published in the same calendar year plus the two years prior.
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Metrics Details
- Citations3
- Citation Indexes3
- CrossRef3
- Captures17
- Readers17
- 17
Article Description
Duchenne's muscular dystrophy (DMD) is a neuromuscular disorder affecting skeletal and cardiac muscle function, caused by mutations in the dystrophin ( DMD ) gene. Dermal fibroblasts, isolated from a DMD patient with a reported deletion of exons 51 to 53 in the DMD gene, were reprogramed into induced pluripotent stem cells (iPSCs) by electroporation with episomal vectors containing the reprograming factors: OCT4, SOX2, LIN28, KLF4, and L-MYC. The obtained iPSC line showed iPSC morphology, expression of pluripotency markers, possessed trilineage differentiation potential and was karyotypically normal.
Bibliographic Details
http://www.sciencedirect.com/science/article/pii/S1873506119301746; http://dx.doi.org/10.1016/j.scr.2019.101544; http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=85073641527&origin=inward; http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=85071166334&origin=inward; http://www.ncbi.nlm.nih.gov/pubmed/31465894; https://linkinghub.elsevier.com/retrieve/pii/S1873506119301746; https://dx.doi.org/10.1016/j.scr.2019.101544
Elsevier BV
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