The Transmembrane Tyrosine Phosphatase DLAR Controls Motor Axon Guidance in Drosophila
Cell, ISSN: 0092-8674, Vol: 84, Issue: 4, Page: 611-622
1996
- 304Citations
- 60Captures
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
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Metrics Details
- Citations304
- Citation Indexes304
- 304
- CrossRef248
- Captures60
- Readers60
- 60
Article Description
DLAR is a receptor-like, transmembrane protein-tyrosine phosphatase in Drosophila that is expressed almost exclusively by developing neurons. Analysis of Dlar loss-of-function mutations shows that DLAR plays a key role during motoneuron growth cone guidance. Segmental nerve b (SNb) motor axons normally exit the common motor pathway, enter the ventral target region, and then synapse on specific ventral muscles. In Dlar mutant embryos, SNb axons bypass their normal target region and instead continue to extend along the common pathway. SNd motor axons also make pathfinding errors, while SNa and SNc axons appear normal. Thus, DLAR controls the ability of certain motor axons to navigate specific choice points in the developing Drosophila nervous system.
Bibliographic Details
http://www.sciencedirect.com/science/article/pii/S0092867400810363; http://dx.doi.org/10.1016/s0092-8674(00)81036-3; http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=0030030320&origin=inward; http://www.ncbi.nlm.nih.gov/pubmed/8598047; http://linkinghub.elsevier.com/retrieve/pii/S0092867400810363; http://api.elsevier.com/content/article/PII:S0092867400810363?httpAccept=text/xml; http://api.elsevier.com/content/article/PII:S0092867400810363?httpAccept=text/plain; https://linkinghub.elsevier.com/retrieve/pii/S0092867400810363; http://dx.doi.org/10.1016/s0092-8674%2800%2981036-3; https://dx.doi.org/10.1016/s0092-8674%2800%2981036-3
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