Molecular mechanisms of ischemic neuronal injury
Annals of Emergency Medicine, ISSN: 0196-0644, Vol: 36, Issue: 5, Page: 483-506
2000
- 210Citations
- 121Captures
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Metrics Details
- Citations210
- Citation Indexes210
- 210
- CrossRef100
- Academic Citation Index (ACI) - airiti2
- Captures121
- Readers121
- 118
Article Description
Brain ischemia triggers a complex cascade of molecular events that unfolds over hours to days. Identified mechanisms of postischemic neuronal injury include altered Ca 2+ homeostasis, free radical formation, mitochondrial dysfunction, protease activation, altered gene expression, and inflammation. Although many of these events are well characterized, our understanding of how they are integrated into the causal pathways of postischemic neuronal death remains incomplete. The primary goal of this review is to provide an overview of molecular injury mechanisms currently believed to be involved in postischemic neuronal death specifically highlighting their time course and potential interactions. [Neumar RW. Molecular mechanisms of ischemic neuronal injury. Ann Emerg Med. November 2000;36:483-506.]
Bibliographic Details
http://www.sciencedirect.com/science/article/pii/S0196064400820284; http://dx.doi.org/10.1067/mem.2000.110995; http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=0033751426&origin=inward; http://www.ncbi.nlm.nih.gov/pubmed/11054204; http://linkinghub.elsevier.com/retrieve/pii/S0196064400820284; http://api.elsevier.com/content/article/PII:S0196064400820284?httpAccept=text/xml; http://api.elsevier.com/content/article/PII:S0196064400820284?httpAccept=text/plain; http://dx.doi.org/10.1016/s0196-0644(00)82028-4; https://linkinghub.elsevier.com/retrieve/pii/S0196064400820284; https://dx.doi.org/10.1067/mem.2000.110995
Elsevier BV
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