Recurrent ischemic cerebrovascular events in a patient with type i antithrombin deficiency caused by 9788 G>A splice site mutation: A case report
Blood Coagulation and Fibrinolysis, ISSN: 0957-5235, Vol: 24, Issue: 2, Page: 213-215
2013
- 8Citations
- 6Captures
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Metrics Details
- Citations8
- Citation Indexes8
- CrossRef4
- Captures6
- Readers6
Article Description
Type I antithrombin deficiency is an autosomal dominant disorder associated with high risk for venous thromboembolism. Data on the association between antithrombin deficiency and arterial thromboembolism are inconsistent. We report here the case of AT deficiency in a 43-year-old man free of cardiovascular risk factors who experienced venous thromboembolism and ischemic stroke followed by two transient ischemic attacks after interruption of oral anticoagulation due to colonoscopy. DNA sequencing of the antithrombin gene revealed heterozygosity for the previously reported substitution G to A at nucleotide position 9788 in intervening sequence four. To our knowledge, this report is the first to show that this genetic abnormality can be associated with recurrent cerebrovascular ischemic events. © 2013 Wolters Kluwer Health Lippincott Williams & Wilkins.
Bibliographic Details
http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=84873363156&origin=inward; http://dx.doi.org/10.1097/mbc.0b013e32835b2467; http://www.ncbi.nlm.nih.gov/pubmed/23358206; https://journals.lww.com/00001721-201303000-00021; http://content.wkhealth.com/linkback/openurl?sid=WKPTLP:landingpage&an=00001721-900000000-99407; https://dx.doi.org/10.1097/mbc.0b013e32835b2467; https://journals.lww.com/bloodcoagulation/toc/9000/00000
Ovid Technologies (Wolters Kluwer Health)
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