Maternal Unresponsiveness and Child Disruptive Problems: The Interplay of Uninhibited Temperament and Dopamine Transporter Genes
Child Development, ISSN: 1467-8624, Vol: 86, Issue: 1, Page: 63-79
2015
- 31Citations
- 121Captures
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Metrics Details
- Citations31
- Citation Indexes31
- 31
- CrossRef26
- Captures121
- Readers121
- 121
Article Description
This study examined how and why dopamine transporter (DAT1) susceptibility alleles moderate the relation between maternal unresponsiveness and young children's behavior problems in a disadvantaged, predominantly minority sample of 201 two-year-old children and their mothers. Using a multimethod, multisource design, the findings indicated that a genetic composite of DAT1 susceptibility alleles (rs27072, rs40184) potentiated associations between maternal unresponsive caregiving and increases in children's behavior problems 2 years later. Moderator-mediated-moderation analyses further revealed that the DAT1 diathesis was more proximally mediated by the potentiating effects of children's uninhibited temperament in the pathway between maternal unresponsiveness and disruptive behavior problems. Results are interpreted in the context of supporting and advancing the biosocial developmental model (Beauchaine & Gatzke-Kopp, 2012).
Bibliographic Details
http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=84922600885&origin=inward; http://dx.doi.org/10.1111/cdev.12281; http://www.ncbi.nlm.nih.gov/pubmed/25130210; https://srcd.onlinelibrary.wiley.com/doi/10.1111/cdev.12281; http://doi.wiley.com/10.1111/cdev.12281; http://onlinelibrary.wiley.com/doi/10.1111/cdev.12281/abstract
Wiley
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