Pathohistological evidence of smoldering inflammation in rheumatic heart disease with massive left atrial calcification
Internal Medicine, ISSN: 1349-7235, Vol: 55, Issue: 7, Page: 751-754
2016
- 8Citations
- 17Captures
Metric Options: CountsSelecting the 1-year or 3-year option will change the metrics count to percentiles, illustrating how an article or review compares to other articles or reviews within the selected time period in the same journal. Selecting the 1-year option compares the metrics against other articles/reviews that were also published in the same calendar year. Selecting the 3-year option compares the metrics against other articles/reviews that were also published in the same calendar year plus the two years prior.
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Metrics Details
- Citations8
- Citation Indexes8
- CrossRef5
- Captures17
- Readers17
- 17
Article Description
A 74-year-old man, who had a history of a mitral valve replacement for rheumatic heart disease (RHD) 30 years previously, was admitted with progressive heart failure. Massive calcification was observed around the left atrium on multidetector CT, in addition to a late gadolinium enhancement (LGE)-positive layer adjacently outside of the calcification on MRI. He underwent a second mitral valve replacement for the prosthetic valve failure. Pathohistological analyses of a tissue section of the left atrial wall from a surgical specimen revealed lymphocyte and macrophage infiltration that coincided with the LGE-positive layer on MRI, suggesting the existence of sustained active inflammation even after the long period of RHD.
Bibliographic Details
http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=84962150449&origin=inward; http://dx.doi.org/10.2169/internalmedicine.55.6125; http://www.ncbi.nlm.nih.gov/pubmed/27041159; https://www.jstage.jst.go.jp/article/internalmedicine/55/7/55_55.6125/_article; https://dx.doi.org/10.2169/internalmedicine.55.6125; https://www.jstage.jst.go.jp/article/internalmedicine/55/7/55_55.6125/_article/-char/en/; https://www.jstage.jst.go.jp/article/internalmedicine/55/7/55_55.6125/_article/-char/ja/
Japanese Society of Internal Medicine
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