Microenvironment-centred dynamics in aggressive b-cell lymphomas
Advances in Hematology, ISSN: 1687-9104, Vol: 2012, Page: 138079
2012
- 17Citations
- 3Usage
- 39Captures
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
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Metrics Details
- Citations17
- Citation Indexes17
- 17
- CrossRef13
- Usage3
- Abstract Views3
- Captures39
- Readers39
- 39
Review Description
Aggressive B-cell lymphomas share high proliferative and invasive attitudes and dismal prognosis despite heterogeneous biological features. In the interchained sequence of events leading to cancer progression, neoplastic clone-intrinsic molecular events play a major role. Nevertheless, microenvironment-related cues have progressively come into focus as true determinants for this process. The cancer-associated microenvironment is a complex network of nonneoplastic immune and stromal cells embedded in extracellular components, giving rise to a multifarious crosstalk with neoplastic cells towards the induction of a supportive milieu. The immunological and stromal microenvironments have been classically regarded as essential partners of indolent lymphomas, while considered mainly negligible in the setting of aggressive B-cell lymphomas that, by their nature, are less reliant on external stimuli. By this paper we try to delineate the cardinal microenvironment-centred dynamics exerting an influence over lymphoid clone progression in aggressive B-cell lymphomas. © 2012 Matilde Cacciatore et al.
Bibliographic Details
http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=84858312362&origin=inward; http://dx.doi.org/10.1155/2012/138079; http://www.ncbi.nlm.nih.gov/pubmed/22400028; http://www.hindawi.com/journals/ah/2012/138079/; https://www.airitilibrary.com/Article/Detail/P20151223001-201212-201707130018-201707130018-16-27; https://dx.doi.org/10.1155/2012/138079; https://www.hindawi.com/journals/ah/2012/138079/
Hindawi Limited
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