Evaluation a renal function of patients with Medication- overuse headache (MOH)
Vol: 9, Issue: 1, Page: 1-10
2022
- 129Usage
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Metrics Details
- Usage129
- Downloads91
- Abstract Views38
Article Description
Excessive drug use causes Medication-overuse headache (MOH) which can be manifested with chronic daily headaches, occurring monthly 15 or more days when the medicament is used redundantly for more than three months. Recent studies concerning the epidemiology of drug- induced disorders suggest that an increased risk of nephrotoxicity appears in a group of patients who abuse NSAIDs. The aim is to confirm the early phase of nephrotoxicity in patients with (MOH), who were treated with NSAIDs in combination with other drugs (analgesics, triptans, and antidepressants) and compared patients treated only with Diclofenac, Piroxicam, Ketoprofen, Paracetamol, Ibuprofen, and Celecoxib. Besides conventional markers of renal functioning (serum/urine creatinine determined by Jaffe methods, enzymatic assay for urea serum). Imunoturbodimetric assay for determination of urinary albumin, microalbuminuria, and β2-microglobulin will be used. Significant glomerular and tubular damage has been reported, and patients on combination therapy with NSAIDs and other drugs (analgesics, triptans, and antidepressants) have seen more glomerular changes than patients treated with NSAID monotherapy.
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