Mechanisms of environmental chemicals that enable the cancer hallmark of evasion of growth suppression
Carcinogenesis, ISSN: 1460-2180, Vol: 36, Issue: Suppl 1, Page: S2-S18
2015
- 57Citations
- 16Usage
- 142Captures
Metric Options: CountsSelecting the 1-year or 3-year option will change the metrics count to percentiles, illustrating how an article or review compares to other articles or reviews within the selected time period in the same journal. Selecting the 1-year option compares the metrics against other articles/reviews that were also published in the same calendar year. Selecting the 3-year option compares the metrics against other articles/reviews that were also published in the same calendar year plus the two years prior.
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
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Metrics Details
- Citations57
- Citation Indexes55
- 55
- CrossRef21
- Clinical Citations1
- 1
- Policy Citations1
- 1
- Usage16
- Abstract Views16
- Captures142
- Readers142
- 142
Review Description
As part of the Halifax Project, this review brings attention to the potential effects of environmental chemicals on important molecular and cellular regulators of the cancer hallmark of evading growth suppression. Specifically, we review the mechanisms by which cancer cells escape the growth-inhibitory signals of p53, retinoblastoma protein, transforming growth factor-beta, gap junctions and contact inhibition. We discuss the effects of selected environmental chemicals on these mechanisms of growth inhibition and cross-reference the effects of these chemicals in other classical cancer hallmarks.
Bibliographic Details
http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=84937697813&origin=inward; http://dx.doi.org/10.1093/carcin/bgv028; http://www.ncbi.nlm.nih.gov/pubmed/26106139; https://academic.oup.com/carcin/article-lookup/doi/10.1093/carcin/bgv028; https://rdw.rowan.edu/som_facpub/78; https://rdw.rowan.edu/cgi/viewcontent.cgi?article=1077&context=som_facpub; https://dx.doi.org/10.1093/carcin/bgv028; https://academic.oup.com/carcin/article-abstract/36/Suppl_1/S2/310663?redirectedFrom=fulltext; http://carcin.oxfordjournals.org/content/36/Suppl_1/S2; https://academic.oup.com/carcin/article-pdf/36/Suppl_1/S2/882192/bgv028.pdf; https://academic.oup.com/carcin/article/36/Suppl_1/S2/310663; http://www.carcin.oxfordjournals.org/lookup/doi/10.1093/carcin/bgv028; https://academic.oup.com/carcin; http://carcin.oxfordjournals.org/cgi/doi/10.1093/carcin/bgv028
Oxford University Press (OUP)
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