Borrelia burgdorferi induces a type I interferon response during early stages of disseminated infection in mice.

Citation data:

BMC microbiology, ISSN: 1471-2180, Vol: 16, Issue: 1, Page: 29

Publication Year:
2016
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Repository URL:
https://touroscholar.touro.edu/nymc_fac_pubs/93; https://touroscholar.touro.edu/cgi/viewcontent.cgi?article=1092&context=nymc_fac_pubs
PMID:
26957120
DOI:
10.1186/s12866-016-0644-4
PMCID:
PMC4784397
Author(s):
Petzke, Mary M; Iyer, Radha; Love, Andrea C; Spieler, Zoe; Brooks, Andrew; Schwartz, Ira
Publisher(s):
Springer Nature; Touro Scholar
Tags:
Immunology and Microbiology; Medicine; Lyme disease; Type I interferon; bacterial pathogenesis; mouse model; Animals; Borrelia burgdorferi; Disease Models, Animal; Female; Humans; Interferon Type I; Lyme Disease; Male; Mice; Mice, Inbred C3H; Bacteriology; Microbiology
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article description
Lyme borrelia genotypes differ in their capacity to cause disseminated disease. Gene array analysis was employed to profile the host transcriptome induced by Borrelia burgdorferi strains with different capacities for causing disseminated disease in the blood of C3H/HeJ mice during early infection.