Elevated level of pro-inflammatory eicosanoids and EPC dysfunction in diabetic patients with cardiac ischemia.

Citation data:

Prostaglandins & other lipid mediators, ISSN: 1098-8823, Vol: 100-101, Issue: 1, Page: 15-21

Publication Year:
2013
Usage 23
Abstract Views 23
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Citations 13
Citation Indexes 13
PMID:
23291334
DOI:
10.1016/j.prostaglandins.2012.12.002
PMCID:
PMC4292900
Author(s):
Issan, Yossi, Hochhauser, Edith, Guo, Austin, Gotlinger, Katherine H, Kornowski, Ran, Leshem-Lev, Dorit, Lev, Eli, Porat, Eyal, Snir, Eitan, Thompson, Carl I, Abraham, Nader G, Laniado-Schwartzman, Michal Show More Hide
Publisher(s):
Elsevier BV
Tags:
Biochemistry, Genetics and Molecular Biology, Pharmacology, Toxicology and Pharmaceutics
article description
Circulating endothelial progenitor cells (EPCs) are recruited from the blood system to sites of ischemia and endothelial damage, where they contribute to the repair and development of blood vessels. Since numerous eicosanoids including leukotrienes (LTs) and hydroxyeicosatetraenoic acids (HETEs) have been shown to exert potent pro-inflammatory activities, we examined their levels in chronic diabetic patients with severe cardiac ischemia in conjunction with the level and function of EPCs.

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