Dysfunction of the ubiquitin/proteasome system and mitochondria in neurodegenerative disease
Mitochondrial Dysfunction in Neurodegenerative Disorders, Page: 141-155
2012
Metric Options: CountsSelecting the 1-year or 3-year option will change the metrics count to percentiles, illustrating how an article or review compares to other articles or reviews within the selected time period in the same journal. Selecting the 1-year option compares the metrics against other articles/reviews that were also published in the same calendar year. Selecting the 3-year option compares the metrics against other articles/reviews that were also published in the same calendar year plus the two years prior.
Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Book Chapter Description
As membranous organelles, mitochondria have been historically subject to a conceptual isolation from the ubiquitin/proteasome system. The degradation of damaged mitochondria is primarily accomplished by macroautophagy, but it has become apparent that the two major proteolytic systems are connected by shared components that are reliant on common molecular pools. The interconnectivity of these systems has implications on neurodegenerative disease.
Bibliographic Details
http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=84948143690&origin=inward; http://dx.doi.org/10.1007/978-0-85729-701-3_9; http://link.springer.com/10.1007/978-0-85729-701-3_9; http://www.springerlink.com/index/10.1007/978-0-85729-701-3_9; http://www.springerlink.com/index/pdf/10.1007/978-0-85729-701-3_9; https://dx.doi.org/10.1007/978-0-85729-701-3_9; https://link.springer.com/chapter/10.1007/978-0-85729-701-3_9
Springer Science and Business Media LLC
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