Immunologic mechanisms in lung carcinogenesis and metastasis
Lung Cancer Metastasis: Novel Biological Mechanisms and Impact on Clinical Practice, Page: 111-134
2009
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Metric Options: CountsSelecting the 1-year or 3-year option will change the metrics count to percentiles, illustrating how an article or review compares to other articles or reviews within the selected time period in the same journal. Selecting the 1-year option compares the metrics against other articles/reviews that were also published in the same calendar year. Selecting the 3-year option compares the metrics against other articles/reviews that were also published in the same calendar year plus the two years prior.
Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Metrics Details
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Book Chapter Description
Progression and metastasis of cancer proceeds in the context of a host response that includes interactions with immune cells that can both attenuate and paradoxically promote the process of metastasis. Growing evidence demonstrating the role of the inflammatory response in carcinogenesis is shedding light on a functional relationship between the host immune system and the malignant neoplasm. The interaction between neoplasm and the immune system can be described with the concepts of (1) cancer immunosurveillance, (2) cancer immunoediting, (3) complicity of the host cellular networks in lung tumorigenesis, and (4) tumor-mediated immunosuppression. Understanding the molecular mechanisms involved in inflammation and lung carcinogenesis provides insight for new drug development that target reversible, non-mutational events in the chemoprevention and treatment of lung cancer. © 2010 Springer-Verlag New York.
Bibliographic Details
http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=84900629802&origin=inward; http://dx.doi.org/10.1007/978-1-4419-0772-1_6; http://link.springer.com/10.1007/978-1-4419-0772-1_6; http://www.springerlink.com/index/10.1007/978-1-4419-0772-1_6; http://www.springerlink.com/index/pdf/10.1007/978-1-4419-0772-1_6; https://dx.doi.org/10.1007/978-1-4419-0772-1_6; https://link.springer.com/chapter/10.1007/978-1-4419-0772-1_6
Springer Science and Business Media LLC
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