Ischemic stroke: Basic pathophysiology and clinical implication
Neuroscience in the 21st Century: From Basic to Clinical, Page: 2543-2563
2013
- 2Citations
- 11Captures
Metric Options: CountsSelecting the 1-year or 3-year option will change the metrics count to percentiles, illustrating how an article or review compares to other articles or reviews within the selected time period in the same journal. Selecting the 1-year option compares the metrics against other articles/reviews that were also published in the same calendar year. Selecting the 3-year option compares the metrics against other articles/reviews that were also published in the same calendar year plus the two years prior.
Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Book Chapter Description
Stroke is one of the most frequent causes of death worldwide. Clinically, stroke is a heterogeneous disease. Brain injury after stroke follows diverse signaling cascades that evolve in a complex spatiotemporal pattern. The core of the infarction is characterized by fast necrotic cell death. In the surrounding area, the penumbra, different cascades lead to delayed forms of cell death. However, endogenous protective mechanisms in the brain are geared to counteract the damaging cascades by mediating ischemic tolerance.
Bibliographic Details
http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=84929283055&origin=inward; http://dx.doi.org/10.1007/978-1-4614-1997-6_97; http://link.springer.com/10.1007/978-1-4614-1997-6_97; http://link.springer.com/content/pdf/10.1007/978-1-4614-1997-6_97; https://dx.doi.org/10.1007/978-1-4614-1997-6_97; https://link.springer.com/referenceworkentry/10.1007/978-1-4614-1997-6_97
Springer Science and Business Media LLC
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