Characterization of plant cell wall damage-associated molecular patterns regulating immune responses
Methods in Molecular Biology, ISSN: 1064-3745, Vol: 1578, Page: 13-23
2017
- 21Citations
- 30Captures
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Metrics Details
- Citations21
- Citation Indexes21
- 21
- CrossRef4
- Captures30
- Readers30
- 30
Book Chapter Description
The plant cell wall is one of the first defensive barriers that pathogens need to overcome to successfully colonize plant tissues. Plant cell wall is considered a dynamic structure that regulates both constitutive and inducible defense mechanisms. The wall is a potential source of a diverse set of Damage-Associated Molecular Patterns (DAMPs), which are signalling molecules that trigger immune responses. However, just a few active wall ligands, such as oligogalacturonic acids (OGs), have been characterized so far. To identify additional wall-derived DAMPs, we obtained different plant wall fractions and tested their capacity to trigger immune responses using a calcium read-out system. To characterize the active DAMPs structures present in these fractions, we applied Glycome Profiling, a technology that uses a large and diverse set of specific monoclonal antibodies against wall carbohydrate ligands. The methods describe here can be used in combination with other biochemical approaches to identify and purify new plant cell wall DAMPs.
Bibliographic Details
http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=85013674835&origin=inward; http://dx.doi.org/10.1007/978-1-4939-6859-6_2; http://www.ncbi.nlm.nih.gov/pubmed/28220412; http://link.springer.com/10.1007/978-1-4939-6859-6_2; https://dx.doi.org/10.1007/978-1-4939-6859-6_2; https://link.springer.com/protocol/10.1007/978-1-4939-6859-6_2
Springer Science and Business Media LLC
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