Kallikrein 8-dependent and independent synaptic tagging and modulation of long-term potentiation: A quest for the associated signaling pathway(s)
Synaptic Tagging and Capture: From Synapses to Behavior: Second Edition, Page: 179-193
2024
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Book Chapter Description
Synaptic tagging is a plausible hypothesis to explain relational memory. However, why and how the tagged synapses can be distinguished from other nonactivated synapses has not yet been clarified. Early-phase long-term potentiation (E-LTP)-related signaling molecules and intracellular molecular trafficking for capturing these toward tagged synapses have been considered essential for the synaptic tagging apparatus. In this chapter, we describe a new mechanism of synaptic tagging that shares the same set of E-LTP induction mechanisms as above; that is, E-LTP-specific proteolysis by Kallikrein 8/neuropsin (KLK8), an extracellular serine protease, is involved in the KLK8-dependent form of synaptic and behavioral tagging.
Bibliographic Details
http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=85204662453&origin=inward; http://dx.doi.org/10.1007/978-3-031-54864-2_9; https://link.springer.com/10.1007/978-3-031-54864-2_9; https://dx.doi.org/10.1007/978-3-031-54864-2_9; https://link.springer.com/chapter/10.1007/978-3-031-54864-2_9
Springer Science and Business Media LLC
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