Animal models of ANCA-associated vasculitides
Systemic Vasculitides: Current Status and Perspectives, Page: 9-13
2016
- 1Citations
- 9Captures
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
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Book Chapter Description
Antibodies against neutrophil proteins myeloperoxidase (MPO) and proteinase- 3 (PR3) are responsible for the development of anti-neutrophil cytoplasmic autoantibodies (ANCA)-associated vasculitides (AAV). Although the knowledge of these conditions is remarkably improved in the last few years, their etiology and pathogenetic mechanism(s) are still poorly understood. The establishment of experimental models has been repeatedly attempted with the aim of achieving a deeper understanding of their human counterpart. Here, we discuss the principal animal models currently used to investigate the mechanisms underlying the onset of AAV.
Bibliographic Details
http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=85018080673&origin=inward; http://dx.doi.org/10.1007/978-3-319-40136-2_2; http://link.springer.com/10.1007/978-3-319-40136-2_2; http://link.springer.com/content/pdf/10.1007/978-3-319-40136-2_2; https://dx.doi.org/10.1007/978-3-319-40136-2_2; https://link.springer.com/chapter/10.1007/978-3-319-40136-2_2
Springer Nature
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