Hyperglycemia and net transintestinal glucose and sodium transport in the rat
Pflügers Archiv European Journal of Physiology, ISSN: 0031-6768, Vol: 390, Issue: 2, Page: 202-206
1981
- 6Citations
Metric Options: CountsSelecting the 1-year or 3-year option will change the metrics count to percentiles, illustrating how an article or review compares to other articles or reviews within the selected time period in the same journal. Selecting the 1-year option compares the metrics against other articles/reviews that were also published in the same calendar year. Selecting the 3-year option compares the metrics against other articles/reviews that were also published in the same calendar year plus the two years prior.
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Metrics Details
- Citations6
- Citation Indexes6
- CrossRef6
Article Description
Hyperglycemia produced by intravenous infusion of glucose causes, in the everted jejunum of rat, an increase of net transintestinal transport of glucose, Na and water without modifying their cell concentration. The concentration of glucose in the fluid pouring out at the serosal side is higher than the calculated intracellular concentration, especially in the experiments in which the serosal compartment is initially empty. This fact could be explained by the possible existence of an active extrusion mechanism in the basolateral membrane of the enterocyte. © 1981 Springer-Verlag.
Bibliographic Details
http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=0019510557&origin=inward; http://dx.doi.org/10.1007/bf00590208; http://www.ncbi.nlm.nih.gov/pubmed/7195569; http://link.springer.com/10.1007/BF00590208; http://www.springerlink.com/index/pdf/10.1007/BF00590208; https://dx.doi.org/10.1007/bf00590208; https://link.springer.com/article/10.1007/BF00590208; http://www.springerlink.com/index/10.1007/BF00590208
Springer Science and Business Media LLC
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