Exogenous carbohydrate oxidation from maltose and glucose ingested during prolonged exercise
European Journal of Applied Physiology and Occupational Physiology, ISSN: 0301-5548, Vol: 64, Issue: 6, Page: 523-527
1992
- 39Citations
- 35Captures
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
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Metrics Details
- Citations39
- Citation Indexes39
- 39
- CrossRef28
- Captures35
- Readers35
- 35
Article Description
Intestinal perfusion studies have shown that glucose absorption from maltose occurs faster than from isocaloric glucose. To determine whether ingested maltose might be a superior source of carbohydrate (CHO) for endurance athletes, we compared the rates of gastric emptying, absorption and oxidation of 15 g · 100 ml solutions of maltose and glucose. Six endurance-trained cyclists drank 1200 ml of either U-C maltose or U-C glucose as a 400-ml loading bolus immediately before exercise, and as 8 × 100-ml drinks at 10-min intervals during a 90-min ride at 700% of maximal oxygen consumption. The rates of gastric emptying [maltose 690 (SD 119) ml · 90 min; glucose 655 (SD 93) ml · 90 min], the appearance of U-C label in the plasma, and the peak rates of exogenous CHO oxidation [maltose 1.0 (SD 0.09) g · min; glucose 0.9 (SD 0.09) g · min] were not significantly different. Further, the 51 (SD 8) g of maltose and the 49 (SD 9) g of glucose oxidised during exercise were similar. Each accounted for approximately 2001o of the total CHO oxidised during the 90 min of exercise. Since only half of the CHO delivered to the intestine was oxidised in the 90-min ride (maltose 49%; glucose 50%), we conclude that neither the rate of gastric emptying, nor digestion limited the rate of ingested CHO utilisation during the early stages of exercise. Rather, we hypothesise that, initially, it could be the rate at which the CHO diffuses across the unstirred water layer of the brush-border of the intestinal villi that limits the utilisation of soluble CHO ingested during prolonged exercise. © 1992 Springer-Verlag.
Bibliographic Details
http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=0026598510&origin=inward; http://dx.doi.org/10.1007/bf00843762; http://www.ncbi.nlm.nih.gov/pubmed/1618190; http://link.springer.com/10.1007/BF00843762; http://www.springerlink.com/index/pdf/10.1007/BF00843762; https://dx.doi.org/10.1007/bf00843762; https://link.springer.com/article/10.1007/BF00843762; http://www.springerlink.com/index/10.1007/BF00843762
Springer Science and Business Media LLC
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