Adoptive-transfer effects of intravenous immunoglobulin in autoimmunity
Journal of Clinical Immunology, ISSN: 0271-9142, Vol: 30, Issue: SUPPL. 1, Page: S20-3
2010
- 10Citations
- 25Captures
Metric Options: CountsSelecting the 1-year or 3-year option will change the metrics count to percentiles, illustrating how an article or review compares to other articles or reviews within the selected time period in the same journal. Selecting the 1-year option compares the metrics against other articles/reviews that were also published in the same calendar year. Selecting the 3-year option compares the metrics against other articles/reviews that were also published in the same calendar year plus the two years prior.
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Metrics Details
- Citations10
- Citation Indexes10
- 10
- CrossRef9
- Captures25
- Readers25
- 25
Review Description
Intravenous immunoglobulin (IVIG) is an effective treatment for a variety of autoimmune and inflammatory conditions. The mechanism of action of IVIG remains poorly understood, but a variety of theories have been suggested. Recent studies in murine models have indicated that some of the ameliorative effects of IVIG in autoimmunity can be repeated by the adoptive transfer of leukocytes that have been primed with IVIG. The active cell component within the leukocyte cell population in immune thrombocytopenia (ITP) was determined to be CD11c+ dendritic cells. This review will highlight recent work in murine systems that indicates that the effects of IVIG can be adoptively transferred in some autoimmune diseases and inflammatory states. © Springer Science+Business Media, LLC 2010.
Bibliographic Details
http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=77956186369&origin=inward; http://dx.doi.org/10.1007/s10875-010-9410-9; http://www.ncbi.nlm.nih.gov/pubmed/20401687; http://link.springer.com/10.1007/s10875-010-9410-9; https://dx.doi.org/10.1007/s10875-010-9410-9; https://link.springer.com/article/10.1007/s10875-010-9410-9; http://www.springerlink.com/index/10.1007/s10875-010-9410-9; http://www.springerlink.com/index/pdf/10.1007/s10875-010-9410-9
Springer Science and Business Media LLC
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