Postmenopausal hypertension
Current Hypertension Reports, ISSN: 1534-3111, Vol: 2, Issue: 2, Page: 202-207
2000
- 39Citations
- 20Captures
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Metrics Details
- Citations39
- Citation Indexes39
- 39
- CrossRef29
- Captures20
- Readers20
- 20
Article Description
Cardiovascular disease is the leading cause of death in women and claims the lives of more than half a million women every year. Hypertension is one of the most prevalent and powerful contributors to atherosclerotic cardiovascular disease. Hypertension affects more men than women until 55 years of age, but after age 55, the percentage of women is higher. Estrogen deficiency has been linked to the rapid increase in cardiovascular disease in women who have undergone natural or surgical menopause. Hormone replacement therapy (HRT) has been shown to decrease the incidence of cardiovascular disease and, in some studies, to reduce blood pressure in postmenopausal women. However, little information is available on the effects of HRT on blood pressure in hypertensive postmenopausal patients. The cardioprotective effects of estrogens are not completely understood but may involve direct effects on blood vessels through modulation of endogenous vasoconstrictors and vasodilators and through reductions in serum lipoprotein and cholesterol levels. Experimental evidence suggests that estrogen increases the biological actions of nitric oxide and decreases the actions of angiotensin. After menopause, loss of the vascular protective effects of estrogens may unmask a population of women particularly prone to hypertension who would be at higher risk for cardiovascular disease. Copyright © 2000 by Current Science Inc.
Bibliographic Details
http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=0034166363&origin=inward; http://dx.doi.org/10.1007/s11906-000-0083-2; http://www.ncbi.nlm.nih.gov/pubmed/10981150; http://link.springer.com/10.1007/s11906-000-0083-2; http://www.springerlink.com/index/10.1007/s11906-000-0083-2; http://www.springerlink.com/index/pdf/10.1007/s11906-000-0083-2; https://dx.doi.org/10.1007/s11906-000-0083-2; https://link.springer.com/article/10.1007/s11906-000-0083-2
Springer Science and Business Media LLC
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