Atherosclerosis regression
Current Treatment Options in Cardiovascular Medicine, ISSN: 1092-8464, Vol: 10, Issue: 3, Page: 187-194
2008
- 1Citations
- 20Captures
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Review Description
Atherosclerosis follows the deposition, retention, and oxidative modification of lipoproteins, especially low-density lipoprotein (LDL) in the walls of large arteries. Uptake of oxidized LDL results in the formation of macrophage foam cells. Proliferation of vascular smooth muscle cells and secretion of extracellular matrix contribute "fibrous" components of the plaque, whereas ongoing accumulation of lipid and inflammatory cell debris forms the necrotic lipid core of the mature atherosclerotic plaque. Both the size and composition of plaques determine the clinical course. In particular, a large lipid core, thin fibrous cap, dense inflammatory cell infiltrate, and proteolytic enzyme activity are associated with adverse risk. Atherosclerosis has often been considered a relentlessly progressive disease. However, new imaging techniques that can quantify plaque burden and provide insights into some of the specific plaque components have allowed regression to be mapped for the first time. In this article, drugs targeting atherosclerosis that have potential or proven benefit in atherosclerosis regression are discussed. Copyright © 2008 by Current Medicine Group LLC.
Bibliographic Details
http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=56349149981&origin=inward; http://dx.doi.org/10.1007/s11936-008-0020-2; http://www.ncbi.nlm.nih.gov/pubmed/18582407; http://link.springer.com/10.1007/s11936-008-0020-2; http://www.springerlink.com/index/10.1007/s11936-008-0020-2; http://www.springerlink.com/index/pdf/10.1007/s11936-008-0020-2; https://dx.doi.org/10.1007/s11936-008-0020-2; https://link.springer.com/article/10.1007/s11936-008-0020-2
Springer Science and Business Media LLC
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