Somatostatin is not co-localized in cholinergic neurons innervating the rat cerebral cortex-hippocampal formation
Brain Research, ISSN: 0006-8993, Vol: 243, Issue: 1, Page: 169-172
1982
- 56Citations
- 2Captures
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Metrics Details
- Citations56
- Citation Indexes56
- CrossRef56
- 53
- Captures2
- Readers2
Article Description
The somatostatin content and choline acetyltransferase activity of the rat cerebral cortex and hippocampus were examined after lesions to the nucleus basalis, the fornix and the dorsal hippocampus. Lesions of the nucleus basalis caused reductions in cortical choline acetyltransferase activity but had no effect on the concentration of somatostatin. Fornix transection caused a reduction in choline acetyltransferase activity in the dorsal hippocampus, but again was without effect on the somatostatin content. Excitotoxin lesions of the dorsal hippocampus caused a 69% reduction in somatostatin concentration in this structure, with no reduction in choline acetyltransferase activity. The results indicate that somatostatin and choline acetyltransferase are in separate populations of neurons in the rat cerebral cortex and hippocampus.
Bibliographic Details
http://www.sciencedirect.com/science/article/pii/0006899382911337; http://dx.doi.org/10.1016/0006-8993(82)91133-7; http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=0019952437&origin=inward; http://www.ncbi.nlm.nih.gov/pubmed/6126255; https://linkinghub.elsevier.com/retrieve/pii/0006899382911337; http://dx.doi.org/10.1016/0006-8993%2882%2991133-7; https://dx.doi.org/10.1016/0006-8993%2882%2991133-7
Elsevier BV
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