Immunocytochemical evidence for direct connections between neurotensin-containing axons and dopaminergic neurons in the rat ventral midbrain tegmentum
Brain Research, ISSN: 0006-8993, Vol: 479, Issue: 2, Page: 402-406
1989
- 59Citations
- 10Captures
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Metrics Details
- Citations59
- Citation Indexes59
- 59
- CrossRef54
- Captures10
- Readers10
- 10
Article Description
A light- and electron-microscopic double antigen localization technique was employed to study cellular relationships between neurotensin (NT)-containing axons and dopaminergic neurons in the substantia nigra and ventral tegmental area of the rat. Direct contacts were observed between NT-immunoreactive axon terminals and the dendrites and perikarya of neurons containing the dopamine biosynthetic enzyme, tyrosine hydroxylase (TH). NT-positive terminals were also found to contact unlabeled dendrites and neuronal somata. These results reveal an ultrastructural substrate through which endogenous NT might influence the activity of dopaminergic neurons in the ventral midbrain tegmentum.
Bibliographic Details
http://www.sciencedirect.com/science/article/pii/0006899389916491; http://dx.doi.org/10.1016/0006-8993(89)91649-1; http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=0024492556&origin=inward; http://www.ncbi.nlm.nih.gov/pubmed/2564307; https://linkinghub.elsevier.com/retrieve/pii/0006899389916491; http://dx.doi.org/10.1016/0006-8993%2889%2991649-1; https://dx.doi.org/10.1016/0006-8993%2889%2991649-1
Elsevier BV
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