Lipids in cells of atherosclerotic and uninvolved human aorta
Experimental and Molecular Pathology, ISSN: 0014-4800, Vol: 42, Issue: 1, Page: 117-137
1985
- 94Citations
- 6Captures
Metric Options: CountsSelecting the 1-year or 3-year option will change the metrics count to percentiles, illustrating how an article or review compares to other articles or reviews within the selected time period in the same journal. Selecting the 1-year option compares the metrics against other articles/reviews that were also published in the same calendar year. Selecting the 3-year option compares the metrics against other articles/reviews that were also published in the same calendar year plus the two years prior.
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Metrics Details
- Citations94
- Citation Indexes94
- 94
- CrossRef79
- Captures6
- Readers6
Article Description
Phospholipid, triglyceride, cholesterol, and cholesteryl ester contents were measured in unaffected and atherosclerotic areas of human aorta and in a suspension of enzyme-isolated cells from these segments. Aortic tissue and the cells isolated from it, as well as intimal and medial cells, significantly differ in lipid content. As lipoidosis develops in an atherosclerotic lesion, lipids accumulate unevenly in the tissue and cells. In zones of fatty infiltration, lipids accumulate, apparently, mainly inside cells while in the fatty streak and atherosclerotic plaque they predominate in the extracellular space. In a suspension of cells derived from both an atherosclerotic lesion and the underlying media, cholesteryl esters are the main component of excessive fat. In the primary culture of cells enzyme-isolated from unaffected intima, fatty streak, and plaque, the lipid content and composition are retained until Days 12 to 14 and are similar to those of freshly isolated cells.
Bibliographic Details
http://www.sciencedirect.com/science/article/pii/001448008590022X; http://dx.doi.org/10.1016/0014-4800(85)90022-x; http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=0021962809&origin=inward; http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=0021998517&origin=inward; http://www.ncbi.nlm.nih.gov/pubmed/3967748; http://linkinghub.elsevier.com/retrieve/pii/001448008590022X; http://api.elsevier.com/content/article/PII:001448008590022X?httpAccept=text/xml; http://api.elsevier.com/content/article/PII:001448008590022X?httpAccept=text/plain; https://linkinghub.elsevier.com/retrieve/pii/001448008590022X; http://dx.doi.org/10.1016/0014-4800%2885%2990022-x; https://dx.doi.org/10.1016/0014-4800%2885%2990022-x; http://www.sciencedirect.com/science/article/pii/001448008590022X?via%3Dihub
Elsevier BV
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