Double-strand break repair and G 2 block in Chinese hamster ovary cells and their radiosensitive mutants
Mutation Research/DNA Repair Reports, ISSN: 0167-8817, Vol: 145, Issue: 3, Page: 177-183
1985
- 82Citations
- 5Captures
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Metrics Details
- Citations82
- Citation Indexes82
- 82
- CrossRef47
- Captures5
- Readers5
Article Description
Two X-ray-sensitive mutants of the CHO K1 cell line were examined for their cell-cycle progression after irradiation with γ-rays, and for their ability to rejoin double-strand breaks (DSBs) as detected by neutral filter elution. Both mutants were impaired in DSB rejoining and both were irreversibly blocked in the G 2 phase of the cell cycle as determined by cytofluorometry. From one mutant we have isolated several revertants. The revertants stem from genomic DNA transfection experiments and may have been caused by gene uptake. All revertants survived γ-irradiation as did the wild-type CHO line. One of them has been examined for its ability to rejoin DSBs and was found to be similar to the wild type.
Bibliographic Details
http://www.sciencedirect.com/science/article/pii/0167881785900252; http://dx.doi.org/10.1016/0167-8817(85)90025-2; http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=0021979299&origin=inward; http://www.ncbi.nlm.nih.gov/pubmed/4039032; https://linkinghub.elsevier.com/retrieve/pii/0167881785900252; http://dx.doi.org/10.1016/0167-8817%2885%2990025-2; https://dx.doi.org/10.1016/0167-8817%2885%2990025-2
Elsevier BV
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