Plasticity, genetics and epigenetics in dystonia: An update
Handbook of Clinical Neurology, ISSN: 0072-9752, Vol: 184, Page: 199-206
2022
- 4Citations
- 8Captures
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Metrics Details
- Citations4
- Citation Indexes4
- Captures8
- Readers8
Book Chapter Description
Dystonia represents a group of movement disorders characterized by involuntary muscle contractions that result in abnormal posture and twisting movements. In the last 20 years several animal models have been generated, greatly improving our knowledge of the neural and molecular mechanism underlying this pathological condition, but the pathophysiology remains still poorly understood. In this review we will discuss recent genetic factors related to dystonia and the current understanding of synaptic plasticity alterations reported by both clinical and experimental research. We will also present recent evidence involving epigenetics mechanisms in dystonia.
Bibliographic Details
http://www.sciencedirect.com/science/article/pii/B9780128194102000114; http://dx.doi.org/10.1016/b978-0-12-819410-2.00011-4; http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=85122822226&origin=inward; http://www.ncbi.nlm.nih.gov/pubmed/35034734; https://linkinghub.elsevier.com/retrieve/pii/B9780128194102000114; https://zenodo.org/record/6258406; https://dx.doi.org/10.1016/b978-0-12-819410-2.00011-4
Elsevier BV
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