The correlation among QTc interval, hyperglycaemia and the impaired autonomic activity
Autonomic Neuroscience, ISSN: 1566-0702, Vol: 154, Issue: 1, Page: 94-98
2010
- 29Citations
- 29Captures
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
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Metrics Details
- Citations29
- Citation Indexes29
- 29
- CrossRef19
- Captures29
- Readers29
- 29
Article Description
The QT interval reflects the duration of the intracellular action potential. Little is known on the interval QT duration in non diabetic insulin-resistant subjects. The aims of the current study were to evaluate the QTc interval in three groups of non diabetic insulin-resistant subjects and the possible correlation between QTc and the autonomic nervous system's activity. 90 subjects were divided in subjects with impaired fasting glycaemia (IFG) and, by the results of OGTT, according to the criteria of ADA, in subjects with normal glucose regulation (NGR) and impaired glucose tolerance (IGT). Insulin resistance was evaluated by the homeostasis model assessment-index (HOMA-I). Heart rate variability and Qtc were calculated. QTc interval is correlated with SDNN, LF n.u. and LF/HF. We have observed that the QTc interval is prolonged in insulin-resistant subjects with associated impaired glucose metabolism, while no difference was reported between insulin-resistant and non insulin-resistant subjects with normal glucose regulation. We hypothesize that hyperglycaemia could play a major role than hyperinsulinemia on the QTc prolongation.
Bibliographic Details
http://www.sciencedirect.com/science/article/pii/S1566070209005670; http://dx.doi.org/10.1016/j.autneu.2009.11.006; http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=77949541582&origin=inward; http://www.ncbi.nlm.nih.gov/pubmed/19963442; https://linkinghub.elsevier.com/retrieve/pii/S1566070209005670; https://dx.doi.org/10.1016/j.autneu.2009.11.006
Elsevier BV
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