Developmental program of mouse Vα14i NKT cells
Current Opinion in Immunology, ISSN: 0952-7915, Vol: 17, Issue: 2, Page: 122-130
2005
- 72Citations
- 27Captures
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
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Metrics Details
- Citations72
- Citation Indexes71
- 71
- CrossRef69
- Patent Family Citations1
- Patent Families1
- Captures27
- Readers27
- 27
Review Description
Natural killer T (NKT) cells are a distinct lymphocyte lineage that regulates immune responses. During their development in the thymus, immature uncommitted double-positive CD4 + CD8 + thymocytes that rearrange the semi-invariant T-cell receptor found on mature NKT cells are positively selected by the non-classical MHC class I molecule CD1d, which is expressed at the surface of cortical thymocytes. At this stage, the positively selected cells branch off from the conventional T-cell developmental program and start to acquire activated and/or memory markers and several ‘bona fide’ NK cell attributes. Recent work has started to reveal the specific developmental requirements for this divergent pathway of differentiation. These include several signal transduction molecules, transcription factors and cytokines, including T-bet, members of the NF-κB family, Fyn and IL-15.
Bibliographic Details
http://www.sciencedirect.com/science/article/pii/S095279150500004X; http://dx.doi.org/10.1016/j.coi.2005.01.002; http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=14844303459&origin=inward; http://www.ncbi.nlm.nih.gov/pubmed/15766670; https://linkinghub.elsevier.com/retrieve/pii/S095279150500004X; https://dx.doi.org/10.1016/j.coi.2005.01.002
Elsevier BV
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