Mechanism of mitotic recombination: insights from C. elegans
Current Opinion in Genetics & Development, ISSN: 0959-437X, Vol: 71, Page: 10-18
2021
- 6Citations
- 16Captures
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
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Metrics Details
- Citations6
- Citation Indexes6
- CrossRef1
- Captures16
- Readers16
- 16
Review Description
Homologous recombination (HR) plays a critical role in largely error-free repair of mitotic and meiotic DNA double-strand breaks (DSBs). DSBs are one of the most deleterious DNA lesions, which are repaired by non-homologous end joining (NHEJ), homologous recombination (HR) or, if compromised, micro-homology mediated end joining (MMEJ). If left unrepaired, DSBs can lead to cell death or if repaired incorrectly can result in chromosome rearrangements that drive cancer development. Here, we describe recent advances in the field of mitotic HR made using Caenorhabditis elegans roundworm, as a model system.
Bibliographic Details
http://www.sciencedirect.com/science/article/pii/S0959437X21000757; http://dx.doi.org/10.1016/j.gde.2021.06.005; http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=85109456322&origin=inward; http://www.ncbi.nlm.nih.gov/pubmed/34186335; https://linkinghub.elsevier.com/retrieve/pii/S0959437X21000757; https://dx.doi.org/10.1016/j.gde.2021.06.005
Elsevier BV
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