Advances in the research on myokine-driven regulation of bone metabolism
Heliyon, ISSN: 2405-8440, Vol: 10, Issue: 1, Page: e22547
2024
- 7Citations
- 12Captures
- 3Mentions
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Metrics Details
- Citations7
- Citation Indexes7
- CrossRef7
- Captures12
- Readers12
- 12
- Mentions3
- News Mentions3
- News3
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Review Description
The traditional view posits that bones and muscles interact primarily through mechanical coupling. However, recent studies have revealed that myokines, proteins secreted by skeletal muscle cells, play a crucial role in the regulation of bone metabolism. Myokines are widely involved in bone metabolism, influencing bone resorption and formation by interacting with factors related to bone cell secretion or influencing bone metabolic pathways. Here, we review the research progress on the myokine regulation of bone metabolism, discuss the mechanism of myokine regulation of bone metabolism, explore the pathophysiological relationship between sarcopenia and osteoporosis, and provide future perspectives on myokine research, with the aim of identify potential specific diagnostic markers and therapeutic entry points.
Bibliographic Details
http://www.sciencedirect.com/science/article/pii/S2405844023097554; http://dx.doi.org/10.1016/j.heliyon.2023.e22547; http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=85181820496&origin=inward; http://www.ncbi.nlm.nih.gov/pubmed/38226270; https://linkinghub.elsevier.com/retrieve/pii/S2405844023097554; https://dx.doi.org/10.1016/j.heliyon.2023.e22547
Elsevier BV
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