Increased interleukin-13 levels in patients with chronic heart failure
International Journal of Cardiology, ISSN: 0167-5273, Vol: 131, Issue: 3, Page: 421-423
2009
- 27Citations
- 9Captures
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
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Metrics Details
- Citations27
- Citation Indexes27
- 27
- CrossRef22
- Captures9
- Readers9
Article Description
A great number of basic and clinical studies have demonstrated that inflammatory cytokines play an important role in development and progress of heart failure. However, there is limited information about allergic cytokine interleukin-13 (IL-13). The inflammatory responses mediated by allergic cytokines can cause significant morbidity and mortality when they become chronic. Therefore, we elucidated the role of IL-13 in the pathophysiology of chronic heart failure. We measured plasma IL-13 levels by enzyme-linked immunosorbent assay in 110 patients with chronic heart failure and 20 control subjects. Plasma IL-13 levels were increased in heart failure patients, compared with the controls, in association with NYHA functional class. In addition, IL-13 levels were correlated positively with plasma levels of brain natriuretic peptide and C-reactive protein, and negatively with left ventricular ejection fraction. Plasma IL-13 levels may be useful for evaluating disease severity in chronic heart failure.
Bibliographic Details
http://www.sciencedirect.com/science/article/pii/S0167527307017159; http://dx.doi.org/10.1016/j.ijcard.2007.07.128; http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=58149201061&origin=inward; http://www.ncbi.nlm.nih.gov/pubmed/18031842; https://linkinghub.elsevier.com/retrieve/pii/S0167527307017159; https://dx.doi.org/10.1016/j.ijcard.2007.07.128
Elsevier BV
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