New aspects in the regulation of human B cell functions by complement receptors CR1, CR2, CR3 and CR4
Immunology Letters, ISSN: 0165-2478, Vol: 237, Page: 42-57
2021
- 31Citations
- 47Captures
- 2Mentions
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
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Metrics Details
- Citations31
- Citation Indexes31
- 31
- CrossRef17
- Captures47
- Readers47
- 47
- Mentions2
- News Mentions2
- News2
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Review Description
The involvement of complement in the regulation of antibody responses has been known for long. By now several additional B cell functions – including cytokine production and antigen presentation – have also been shown to be regulated by complement proteins. Most of these important activities are mediated by receptors interacting with activation fragments of the central component of the complement system C3, such as C3b, iC3b and C3d, which are covalently attached to antigens and immune complexes. This review summarizes the role of complement receptors interacting with these ligands, namely CR1 (CD35), CR2 (CD21), CR3 (CD11b/CD18) and CR4 (CD11c/CD18) expressed by B cells in health and disease. Although we focus on human B lymphocytes, we also aim to call the attention to important differences between human and mouse systems.
Bibliographic Details
http://www.sciencedirect.com/science/article/pii/S0165247821001000; http://dx.doi.org/10.1016/j.imlet.2021.06.006; http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=85110676768&origin=inward; http://www.ncbi.nlm.nih.gov/pubmed/34186155; https://linkinghub.elsevier.com/retrieve/pii/S0165247821001000; https://dx.doi.org/10.1016/j.imlet.2021.06.006
Elsevier BV
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