Apelin-13, a regulator of autophagy, apoptosis and inflammation in multifaceted bone protection
International Immunopharmacology, ISSN: 1567-5769, Vol: 117, Page: 109991
2023
- 17Citations
- 7Captures
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Metrics Details
- Citations17
- Citation Indexes17
- 17
- CrossRef10
- Captures7
- Readers7
Review Description
Apelin/APJ is widely distributed in various tissues in the body and participates in the regulation of physiological and pathological mechanisms such as autophagy, apoptosis, inflammation, and oxidative stress. Apelin-13 is an adipokine family member with multiple biological roles and has been shown to be involved in the development and progression of bone diseases. In the process of osteoporosis and fracture healing, Apelin-13 plays an osteoprotective role by regulating the autophagy and apoptosis of BMSCs, and promotes the osteogenic differentiation of BMSCs. In addition, Apelin-13 also attenuates the progression of arthritis by regulating the inflammatory response of macrophages. In conclusion, Apelin-13 has an important connection with bone protection, which provides a new strategy for the clinical treatment of bone-related diseases.
Bibliographic Details
http://www.sciencedirect.com/science/article/pii/S1567576923003120; http://dx.doi.org/10.1016/j.intimp.2023.109991; http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=85150860394&origin=inward; http://www.ncbi.nlm.nih.gov/pubmed/37012875; https://linkinghub.elsevier.com/retrieve/pii/S1567576923003120; https://dx.doi.org/10.1016/j.intimp.2023.109991
Elsevier BV
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