Urban Particulate Matter Air Pollution Is Associated With Subclinical Atherosclerosis
Journal of the American College of Cardiology, ISSN: 0735-1097, Vol: 56, Issue: 22, Page: 1803-1808
2010
- 165Citations
- 152Captures
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Metrics Details
- Citations165
- Citation Indexes155
- 155
- CrossRef129
- Policy Citations10
- Policy Citation10
- Captures152
- Readers152
- 142
Article Description
The aim of this study was to investigate the association of long-term residential exposure to fine particles with carotid intima-media thickness (CIMT). Experimental and epidemiological evidence suggest that long-term exposure to air pollution might have a causal role in atherogenesis, but epidemiological findings are still inconsistent. We investigate whether urban particulate matter (PM) air pollution is associated with CIMT, a marker of subclinical atherosclerosis. We used baseline data (2000 to 2003) from the HNR (Heinz Nixdorf Recall) study, a population-based cohort of 4,814 participants, 45 to 75 years of age. We assessed residential long-term exposure to PM with a chemistry transport model and measured distance to high traffic. Multiple linear regression was used to estimate associations of air pollutants and traffic with CIMT, adjusting for each other, city of residence, age, sex, diabetes, and lifestyle variables. Median CIMT of the 3,380 analyzed participants was 0.66 mm (interquartile range 0.16 mm). An interdecile range increase in PM 2.5 (4.2 μg/m 3 ), PM 10 (6.7 μg/m 3 ), and distance to high traffic (1,939 m) was associated with a 4.3% (95% confidence interval [CI]: 1.9% to 6.7%), 1.7% (95% CI: −0.7% to 4.1%), and 1.2% (95% CI: −0.2% to 2.6%) increase in CIMT, respectively. Our study shows a clear association of long-term exposure to PM 2.5 with atherosclerosis. This finding strengthens the hypothesized role of PM 2.5 as a risk factor for atherogenesis.
Bibliographic Details
http://www.sciencedirect.com/science/article/pii/S0735109710039094; http://dx.doi.org/10.1016/j.jacc.2010.04.065; http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=78449305860&origin=inward; http://www.ncbi.nlm.nih.gov/pubmed/21087707; https://linkinghub.elsevier.com/retrieve/pii/S0735109710039094; https://dx.doi.org/10.1016/j.jacc.2010.04.065
Elsevier BV
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