Association between ApoA-II -265 T/C polymorphism and oxidative stress in patients with type 2 diabetes mellitus
Journal of Diabetes and its Complications, ISSN: 1056-8727, Vol: 29, Issue: 7, Page: 908-912
2015
- 8Citations
- 34Captures
Metric Options: Counts1 Year3 YearSelecting the 1-year or 3-year option will change the metrics count to percentiles, illustrating how an article or review compares to other articles or reviews within the selected time period in the same journal. Selecting the 1-year option compares the metrics against other articles/reviews that were also published in the same calendar year. Selecting the 3-year option compares the metrics against other articles/reviews that were also published in the same calendar year plus the two years prior.
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Metrics Details
- Citations8
- Citation Indexes8
- CrossRef7
- Captures34
- Readers34
- 34
Article Description
Apolipoprotein A-II (ApoA-II) constitutes approximately 20% of the total HDL protein content. The results of various studies on the relationship between cardiovascular diseases (CVD) and the plasma ApoA-II level are contradictory. The aim of this study was to determine the relationship between ApoA-II polymorphism and oxidative stress (OS) as a risk factor for CVD.
Bibliographic Details
http://www.sciencedirect.com/science/article/pii/S1056872715002299; http://dx.doi.org/10.1016/j.jdiacomp.2015.05.024; http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=84939570585&origin=inward; http://www.ncbi.nlm.nih.gov/pubmed/26104730; https://linkinghub.elsevier.com/retrieve/pii/S1056872715002299; http://www.jdcjournal.com/article/S1056-8727(15)00229-9/abstract
Elsevier BV
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