In Utero Exposure to Benzophenone-2 Causes Hypospadias Through an Estrogen Receptor Dependent Mechanism
The Journal of Urology, ISSN: 0022-5347, Vol: 178, Issue: 4, Page: 1637-1642
2007
- 45Citations
- 5Usage
- 33Captures
Metric Options: CountsSelecting the 1-year or 3-year option will change the metrics count to percentiles, illustrating how an article or review compares to other articles or reviews within the selected time period in the same journal. Selecting the 1-year option compares the metrics against other articles/reviews that were also published in the same calendar year. Selecting the 3-year option compares the metrics against other articles/reviews that were also published in the same calendar year plus the two years prior.
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Metrics Details
- Citations45
- Citation Indexes41
- 41
- CrossRef33
- Policy Citations4
- Policy Citation4
- Usage5
- Abstract Views5
- Captures33
- Readers33
- 33
Article Description
Additives such as benzophenone-2 are commonly used in cosmetic products and food container plastics to filter out ultraviolet light. In pregnant women exposure may result in transplacental transfer of benzophenone-2 to fetuses. Benzophenone-2 is estrogenic in vitro and in the rat uterotropic assay. Estradiol causes hypospadias in mice and estrogen-like compounds are also postulated to cause hypospadias. We determined whether hypospadias would develop in male mice exposed to benzophenone-2 in utero and whether this outcome depended on estrogen receptor pathways.
Bibliographic Details
http://www.sciencedirect.com/science/article/pii/S0022534707008889; http://dx.doi.org/10.1016/j.juro.2007.03.190; http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=34548513563&origin=inward; http://www.ncbi.nlm.nih.gov/pubmed/17707034; http://www.jurology.com/doi/10.1016/j.juro.2007.03.190; https://hsrc.himmelfarb.gwu.edu/smhs_uro_facpubs/428; https://hsrc.himmelfarb.gwu.edu/cgi/viewcontent.cgi?article=1428&context=smhs_uro_facpubs; https://dx.doi.org/10.1016/j.juro.2007.03.190; https://www.auajournals.org/doi/10.1016/j.juro.2007.03.190; http://linkinghub.elsevier.com/retrieve/pii/S0022534707008889
Ovid Technologies (Wolters Kluwer Health)
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