Glutamate Receptor Dynamics in Dendritic Microdomains
Neuron, ISSN: 0896-6273, Vol: 58, Issue: 4, Page: 472-497
2008
- 308Citations
- 473Captures
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Metrics Details
- Citations308
- Citation Indexes308
- 308
- CrossRef291
- Captures473
- Readers473
- 473
Review Description
Among diverse factors regulating excitatory synaptic transmission, the abundance of postsynaptic glutamate receptors figures prominently in molecular memory and learning-related synaptic plasticity. To allow for both long-term maintenance of synaptic transmission and acute changes in synaptic strength, the relative rates of glutamate receptor insertion and removal must be tightly regulated. Interactions with scaffolding proteins control the targeting and signaling properties of glutamate receptors within the postsynaptic membrane. In addition, extrasynaptic receptor populations control the equilibrium of receptor exchange at synapses and activate distinct signaling pathways involved in plasticity. Here, we review recent findings that have shaped our current understanding of receptor mobility between synaptic and extrasynaptic compartments at glutamatergic synapses, focusing on AMPA and NMDA receptors. We also examine the cooperative relationship between intracellular trafficking and surface diffusion of glutamate receptors that underlies the expression of learning-related synaptic plasticity.
Bibliographic Details
http://www.sciencedirect.com/science/article/pii/S089662730800408X; http://dx.doi.org/10.1016/j.neuron.2008.04.030; http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=43449102784&origin=inward; http://www.ncbi.nlm.nih.gov/pubmed/18498731; https://linkinghub.elsevier.com/retrieve/pii/S089662730800408X
Elsevier BV
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