Emerging Treatment Strategies for Cerebral Ischemia–Reperfusion Injury
Neuroscience, ISSN: 0306-4522, Vol: 507, Page: 112-124
2022
- 36Citations
- 14Captures
- 2Mentions
Metric Options: Counts1 Year3 YearSelecting the 1-year or 3-year option will change the metrics count to percentiles, illustrating how an article or review compares to other articles or reviews within the selected time period in the same journal. Selecting the 1-year option compares the metrics against other articles/reviews that were also published in the same calendar year. Selecting the 3-year option compares the metrics against other articles/reviews that were also published in the same calendar year plus the two years prior.
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Metrics Details
- Citations36
- Citation Indexes36
- 36
- CrossRef26
- Captures14
- Readers14
- 14
- Mentions2
- News Mentions2
- News2
Most Recent News
Fibroblast Growth Factor 21 Protects Against Cerebral Ischemia/Reperfusion Injury by Inhibiting Oxidative Stress and Ferroptosis
Introduction On a global scale, stroke has emerged as the second major driver of mortality and the third of disability. Over the past thirty years,
Review Description
Cerebral ischemia–reperfusion injury (CI/RI) injury is a common feature of ischemic stroke which occurs when the blood supply is restored after a period of ischemia in the brain. Reduced blood-flow to the brain during CI/RI compromises neuronal cell health as a result of mitochondrial dysfunction, oxidative stress, cytokine production, inflammation and tissue damage. Reperfusion therapy during CI/RI can restore the blood flow to ischemic regions of brain which are not yet infarcted. The long-term goal of CI/RI therapy is to reduce stroke-related neuronal cell death, disability and mortality. A range of drug and interventional therapies have emerged that can alleviate CI/RI mediated oxidative stress, inflammation and apoptosis in the brain. Herein, we review recent studies on CI/RI interventions for which a mechanism of action has been described and the potential of these therapeutic modalities for future use in the clinic.
Bibliographic Details
http://www.sciencedirect.com/science/article/pii/S0306452222005371; http://dx.doi.org/10.1016/j.neuroscience.2022.10.020; http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=85141953055&origin=inward; http://www.ncbi.nlm.nih.gov/pubmed/36341725; https://linkinghub.elsevier.com/retrieve/pii/S0306452222005371; https://dx.doi.org/10.1016/j.neuroscience.2022.10.020
Elsevier BV
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