Histopathology of chronic renal allograft dysfunction
Transplantation Reviews, ISSN: 0955-470X, Vol: 18, Issue: 2, Page: 80-85
2004
- 5Captures
Metric Options: CountsSelecting the 1-year or 3-year option will change the metrics count to percentiles, illustrating how an article or review compares to other articles or reviews within the selected time period in the same journal. Selecting the 1-year option compares the metrics against other articles/reviews that were also published in the same calendar year. Selecting the 3-year option compares the metrics against other articles/reviews that were also published in the same calendar year plus the two years prior.
Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Metrics Details
- Captures5
- Readers5
Article Description
The rate of late allograft loss remains relatively constant, despite greatly improved success in the early management of renal transplants. The pathological changes encountered in kidneys undergoing late allograft dysfunction are the result of a variety of processes, both immune and nonimmune. The gradual appearance of interstitial fibrosis and tubular atrophy is a nonspecific finding, which characterizes late allograft dysfunction. Features that indicate chronic rejection include transplant glomerulopathy, vascular intimal hyperplasia, particularly in association with intimal lymphocytic infiltration. Both of these changes may be related to humoral rejection. Animal models provide important insights into the mechanism of chronic rejection. In a commonly used model, the Fisher to Lewis rat model, antidonor antibodies against matrix proteins are associated with the development of transplant glomerulopathy, and the appearance of antitubular antibodies and a granulomatous interstitial nephritis may indicate graft-host differences in tubular basement membrane structure.
Bibliographic Details
http://www.sciencedirect.com/science/article/pii/S0955470X04000096; http://dx.doi.org/10.1016/j.trre.2004.03.005; http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=3242713461&origin=inward; https://linkinghub.elsevier.com/retrieve/pii/S0955470X04000096; https://api.elsevier.com/content/article/PII:S0955470X04000096?httpAccept=text/xml; https://api.elsevier.com/content/article/PII:S0955470X04000096?httpAccept=text/plain; https://dx.doi.org/10.1016/j.trre.2004.03.005
Elsevier BV
Provide Feedback
Have ideas for a new metric? Would you like to see something else here?Let us know