Regulation of skeletal myogenesis by Notch
Experimental Cell Research, ISSN: 0014-4827, Vol: 316, Issue: 18, Page: 3028-3033
2010
- 93Citations
- 140Captures
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Metrics Details
- Citations93
- Citation Indexes93
- 93
- CrossRef63
- Captures140
- Readers140
- 140
Review Description
Notch signaling has emerged as a key player in skeletal muscle development and regeneration. Simply stated, Notch signaling inhibits differentiation. Accordingly, fine-tuning the pathway is essential for proper muscle homeostasis. This review will address various aspects of Notch signaling, including our current views of the core pathway, its effects in muscle, its interactions with other signaling pathways, and its relationship with ageing.
Bibliographic Details
http://www.sciencedirect.com/science/article/pii/S0014482710002077; http://dx.doi.org/10.1016/j.yexcr.2010.05.002; http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=77957686086&origin=inward; http://www.ncbi.nlm.nih.gov/pubmed/20452344; https://linkinghub.elsevier.com/retrieve/pii/S0014482710002077
Elsevier BV
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