Protective Role of TNF- α in Respiratory Syncytial Virus Infection In Vitro and In Vivo
The American Journal of the Medical Sciences, ISSN: 0002-9629, Vol: 311, Issue: 5, Page: 201-204
1996
- 2Citations
- 7Captures
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Metrics Details
- Citations2
- Citation Indexes2
- CrossRef2
- Captures7
- Readers7
Article Description
Respiratory syncytial virus (RSV) infection causes substantial morbidity in young children and immunocompromised adults, yet its pathogenesis is poorly understood. Because the proinflammatory cytokine tumor necrosis factoralpha (TNF- α ) may be important in host response to viral infection, HEp-2 cells were treated with TNF- α and mice were given TNF- α antibody before RSV infection. Pretreatment of HEp-2 cells with TNF- α inhibited RSV replication as determined by cytopathic effect. Respiratory syncytial virus-infected BALB/c mice treated with antibody to TNF- α had greater maximal weight loss and slower recovery time than control mice. These results suggest a protective role for TNF- α in RSV infection.
Bibliographic Details
http://www.sciencedirect.com/science/article/pii/S0002962915416957; http://dx.doi.org/10.1016/s0002-9629(15)41695-7; https://linkinghub.elsevier.com/retrieve/pii/S0002962915416957; http://linkinghub.elsevier.com/retrieve/pii/S0002962915416957; http://api.elsevier.com/content/article/PII:S0002962915416957?httpAccept=text/xml; http://api.elsevier.com/content/article/PII:S0002962915416957?httpAccept=text/plain; http://dx.doi.org/10.1016/s0002-9629%2815%2941695-7; https://dx.doi.org/10.1016/s0002-9629%2815%2941695-7
Elsevier BV
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