Chronic psychosocial stress-induced down-regulation of immunity depends upon individual factors
Journal of Neuroimmunology, ISSN: 0165-5728, Vol: 141, Issue: 1, Page: 58-64
2003
- 29Citations
- 28Captures
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Metrics Details
- Citations29
- Citation Indexes27
- 27
- CrossRef19
- Policy Citations2
- 2
- Captures28
- Readers28
- 28
Article Description
The effect of chronic stress on immune functions is strongly biased by individual factors. Mice were subjected to a new model of chronic psychosocial stress in which four different subcategories of stressed animals may be identified: Resident Dominants (RD), Resident Subordinates (RS), Intruder Dominants (InD), and Intruder Subordinates (InS). After 7 days of stress, mice were immunized with keyhole limpet hemocyanine (KLH). Their immune functions were investigated 14 days later with stress continuing trough. Importantly, RS mice, which are mice losing territory ownership, were the more affected, having lower IgG, proliferation, and IL-2. RD and InD showed lower IgG while InS showed no immune alteration. In conclusion, loss of resources could be a key factor in determining individual vulnerability to stressful events.
Bibliographic Details
http://www.sciencedirect.com/science/article/pii/S0165572803002200; http://dx.doi.org/10.1016/s0165-5728(03)00220-0; http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=0142107312&origin=inward; http://www.ncbi.nlm.nih.gov/pubmed/12965254; https://linkinghub.elsevier.com/retrieve/pii/S0165572803002200; http://linkinghub.elsevier.com/retrieve/pii/S0165572803002200; http://api.elsevier.com/content/article/PII:S0165572803002200?httpAccept=text/xml; http://api.elsevier.com/content/article/PII:S0165572803002200?httpAccept=text/plain; http://dx.doi.org/10.1016/s0165-5728%2803%2900220-0; https://dx.doi.org/10.1016/s0165-5728%2803%2900220-0
Elsevier BV
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