Japanese encephalitis virus stimulates superoxide dismutase activity in rat glial cultures
Neuroscience Letters, ISSN: 0304-3940, Vol: 324, Issue: 2, Page: 133-136
2002
- 47Citations
- 28Captures
Metric Options: CountsSelecting the 1-year or 3-year option will change the metrics count to percentiles, illustrating how an article or review compares to other articles or reviews within the selected time period in the same journal. Selecting the 1-year option compares the metrics against other articles/reviews that were also published in the same calendar year. Selecting the 3-year option compares the metrics against other articles/reviews that were also published in the same calendar year plus the two years prior.
Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Metrics Details
- Citations47
- Citation Indexes47
- 47
- CrossRef32
- Captures28
- Readers28
- 28
Article Description
Japanese encephalitis virus (JEV) infection is commonly associated with inflammatory reaction and neurological disease that occurs in the infected animals. Reactive oxygen species have been implicated as a critical mediator for inflammation and diseases. The present study investigated the change of redox potential in glial cells following JEV infection. JEV infection induced the generation of superoxide anion and nitric oxide in rat cortical glial cells. Manganese superoxide dismutase, but not copper/zinc superoxide dismutase was activated by JEV infection, and this activation was blocked by pyrrolidine dithiocarbamate. In addition, the increased superoxide dismutase activity was also apparent in JEV acutely, or persistently infected continuous cell lines. These results suggest that cellular factors regulating oxidative pathway might play roles in responding to JEV infection.
Bibliographic Details
http://www.sciencedirect.com/science/article/pii/S0304394002002367; http://dx.doi.org/10.1016/s0304-3940(02)00236-7; http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=0037123727&origin=inward; http://www.ncbi.nlm.nih.gov/pubmed/11988345; https://linkinghub.elsevier.com/retrieve/pii/S0304394002002367; http://dx.doi.org/10.1016/s0304-3940%2802%2900236-7; https://dx.doi.org/10.1016/s0304-3940%2802%2900236-7
Elsevier BV
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