Glycation des HDL et conséquences chez le patient diabétique
Médecine des Maladies Métaboliques, ISSN: 1957-2557, Vol: 7, Issue: 5, Page: 431-436
2013
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Article Description
Les HDL ont un rôle majeur dans l’homéostasie vasculaire. Leur action protectrice passe notamment par leur récepteur spécifique SR-BI (pour scavenger receptor de classe B type 1 ). La glycation des HDL, phénomène majeur observé au cours d’un diabète, a de nombreuses conséquences néfastes vasculaires. La glycation des HDL entraîne une augmentation de leur catabolisme, un défaut du transport inverse du cholestérol, une perte de leurs propriétés anti-inflammatoires et anti-oxydantes, notamment sur les LDL oxydées, lipoprotéines athérogènes présentes en abondance dans les maladies chroniques. Cliniquement, cela se traduit par une dysfonction endothéliale, phénomène cardiovasculaire délétère, observée notamment au cours du diabète. HDLs play a major role in vascular homeostasis. Their protective actions are mediated in part by their SR-BI specific receptor. HDLs glycation, huge phenomenon observed during diabetes mellitus lead to their increased catabolism, a decrease in the reverse transport of cholesterol, a loss of their antiinflammatory and antioxidant properties, particularly on oxidized LDL, abundant lipoparticules proatherogenic observed in chronic diseases. Endothelial dysfunction, a deleterious vascular phenomenon, observed in diabetes, is the clinical representation of these adverse effects of HDL glycation.
Bibliographic Details
http://www.sciencedirect.com/science/article/pii/S1957255713705327; http://dx.doi.org/10.1016/s1957-2557(13)70532-7; http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=84900818960&origin=inward; https://linkinghub.elsevier.com/retrieve/pii/S1957255713705327; http://linkinghub.elsevier.com/retrieve/pii/S1957255713705327; http://api.elsevier.com/content/article/PII:S1957255713705327?httpAccept=text/xml; http://api.elsevier.com/content/article/PII:S1957255713705327?httpAccept=text/plain; http://dx.doi.org/10.1016/s1957-2557%2813%2970532-7; https://dx.doi.org/10.1016/s1957-2557%2813%2970532-7
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