Vascular Calcification in Uremia: What Is New and Where Are We Going?
Advances in Chronic Kidney Disease, ISSN: 1548-5595, Vol: 15, Issue: 4, Page: 413-419
2008
- 21Citations
- 12Captures
Metric Options: CountsSelecting the 1-year or 3-year option will change the metrics count to percentiles, illustrating how an article or review compares to other articles or reviews within the selected time period in the same journal. Selecting the 1-year option compares the metrics against other articles/reviews that were also published in the same calendar year. Selecting the 3-year option compares the metrics against other articles/reviews that were also published in the same calendar year plus the two years prior.
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Metrics Details
- Citations21
- Citation Indexes21
- 21
- CrossRef17
- Captures12
- Readers12
- 12
Article Description
Arterial calcification is very common in patients with chronic kidney disease (CKD) and other chronic inflammatory disorders such as diabetes mellitus. Arterial calcification is associated with significant morbidity and increased early mortality. Vascular calcification is a highly orchestrated process that entrains a repertoire of transcription factors and involves the activation of an osteogenic program that recapitulates the molecular fingerprints seen in bone formation. Recent studies have implicated the inflammatory cytokine tumor necrosis factor α in the pathobiology of arterial calcification. Metabolic acidosis, which is prevalent in patients with advanced kidney disease, has also been shown in some recent studies to attenuate vascular calcification in animal models. In this review, we summarize the recent advances in understanding the molecular mechanisms underpinning vascular mineralization and discuss their implications in terms of translational opportunities, unmet needs, and future directions.
Bibliographic Details
http://www.sciencedirect.com/science/article/pii/S1548559508001146; http://dx.doi.org/10.1053/j.ackd.2008.07.011; http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=52049097061&origin=inward; http://www.ncbi.nlm.nih.gov/pubmed/18805387; https://linkinghub.elsevier.com/retrieve/pii/S1548559508001146
Elsevier BV
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