Gene-environment interaction in coronary artery disease: Apolipoprotein E and smoking and the interleukin-6 gene and inflammation as examples
Coronary Heart Disease Epidemiology: From Aetiology to Public Health, Page: 528-544
2009
- 2Captures
Metric Options: CountsSelecting the 1-year or 3-year option will change the metrics count to percentiles, illustrating how an article or review compares to other articles or reviews within the selected time period in the same journal. Selecting the 1-year option compares the metrics against other articles/reviews that were also published in the same calendar year. Selecting the 3-year option compares the metrics against other articles/reviews that were also published in the same calendar year plus the two years prior.
Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Metrics Details
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Book Chapter Description
This chapter discusses gene-environment interaction in coronary artery disease (CAD). Topics covered include risk factors for CAD, apolipoprotein E and effect on lipid levels and coronary heart disease risk, APOE genotype-smoking interaction, homeostasis and use of stressing the genotype to identify functional variants, and the role of inflammatory processes in atherogenesis.
Bibliographic Details
http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=84921259652&origin=inward; http://dx.doi.org/10.1093/acprof:oso/9780198525738.003.0032; https://academic.oup.com/book/40090/chapter/341137288; http://dx.doi.org/10.1093/acprof%3Aoso/9780198525738.003.0032; https://dx.doi.org/10.1093/acprof%3Aoso/9780198525738.003.0032; https://academic.oup.com/book/40090/chapter-abstract/341137288?redirectedFrom=fulltext
Oxford University Press (OUP)
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