Foxp3 T cells in humoral immunity
International Immunology, ISSN: 0953-8178, Vol: 26, Issue: 2, Page: 61-69
2014
- 73Citations
- 130Captures
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Example: if you select the 1-year option for an article published in 2019 and a metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019. If you select the 3-year option for the same article published in 2019 and the metric category shows 90%, that means that the article or review is performing better than 90% of the other articles/reviews published in that journal in 2019, 2018 and 2017.
Citation Benchmarking is provided by Scopus and SciVal and is different from the metrics context provided by PlumX Metrics.
Metrics Details
- Citations73
- Citation Indexes73
- 73
- CrossRef66
- Captures130
- Readers130
- 130
Article Description
T cells are essential for the maintenance of immune homeostasis and prevention of autoimmunity. In humoral immune responses, loss of T cell function causes increased levels of serum autoantibodies, hyper-IgE, spontaneous generation of germinal centres, and enhanced numbers of specialised T follicular helper cells (T cells) controlled by the lineage-defining transcription factor BCL-6 (B-cell lymphoma 6). Recent studies have demonstrated that a subset of T cells [T follicular regulatory (T) cells] are able to co-opt the follicular T-cell program by gaining expression of BCL-6 and travelling to the follicle where they have an important role in the control of expansion of Tcells and the germinal centre reaction. However, the mechanisms by which they exert this control are still under investigation. In this review, we discuss the effects of T cells on humoral immunity and the mechanisms by which they exert their regulatory function. © The Japanese Society for Immunology 2013. All rights reserved.
Bibliographic Details
Oxford University Press (OUP)
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